[284] Furthermore, the research group also identified that METTL3 N terminal interacts with eIF3 subunit h and control the translation initiation in lung cancers independent of m6A. [285] In gastric cancer, METTL3 has also been found to bind to non-m6A-modified target mRNAs and further promotes their translation by interacting with eIF4F (Fig. 5). Here, EIF3A is linked to lung cancer.