The vitamin D receptor (VDR) upregulated the expression of TOPORS-AS1 while hnRNPA2B1 (heterogeneous nuclear ribonucleoprotein A2B1) directly interacted with TOPORS-AS1, both of which elicit the inhibition of β-catenin and suppress the proliferation of ovarian cancer cells [42]. This evidence concerns the gene HNRNPA2B1 and ovarian cancer.