The vitamin D receptor (VDR) upregulated the expression of TOPORS-AS1 while hnRNPA2B1 (heterogeneous nuclear ribonucleoprotein A2B1) directly interacted with TOPORS-AS1, both of which elicit the inhibition of β-catenin and suppress the proliferation of ovarian cancer cells [42]. Here, VDR is linked to ovarian carcinoma.