NFE2L2 and metabolic dysfunction-associated steatotic liver disease: Indeed, these two compounds, even if different in composition, act as direct radical-scavenging antioxidants modulating different down-stream target pathways, such as nuclear factor erythroid 2-related factor 2-dependent transcriptional regulation of genes that may reduce ROS reactive oxygen species and lipid peroxidation and improve mitochondrial homeostasis, thus decreasing hepato-metabolic impairment associated to MASLD [58,59,60].