In the model group, quantitative analysis revealed significant decreases in cortical BDNF and NT3 levels compared with the control group, emphasizing the low neurotrophic state of neurons in the AD model mice, while SCEP treatment upregulated the levels of BDNF (p > 0.05) and NT3 (p < 0.01 at both the hippocampus and cortex) proteins in the brains of model mice (Figure 8C–F). This evidence concerns the gene BDNF and Alzheimer disease.