NAT10 and esophageal squamous cell carcinoma: By establishing an experimental metastasis mouse model through intravenous injection of ESCC cells into NOD‐Prkdcem26Cd52 Il2rgem26Cd22 (NCG) mice, we observed that NAT10 depletion resulted in a successful delay of lung metastasis indicated by weaker bioluminescence signals, whereas overexpression of ATP6V0E1 recovered the metastatic ability of ESCC cells (Figure 4I,J).