FOXP3 and ulcerative colitis: Its expression profile has been linked to the mitigation of autoimmune responses [32]。Considering the critical role of Treg cells in the pathogenesis of ulcerative colitis, the investigation into FOXP3 holds considerable significance [29,33]。Additionally, STAT6, identified as a pivotal regulator of ferroptosis [34,35], assumes a significant role in the context of ulcerative colitis [36,37].