BIN2 and infection: At the same times, the JA, ABA, and SA f signal pathways were activated in the resistant material by the downregulation of JAZ, ABF, and TGA, respectively, at 2 dpi, while the JA and ABA signal pathways were depressed in the susceptible material by the upregulation of JAZ and PP2C. Notably, two critical BR signaling regulators showed coordinated upregulation at 2 dpi in the susceptible material: BIN2 (GSK3 kinase) and its interacting partner BKI1. This dual induction suggested a strategic BR signaling defense–growth integrator during early infection.