Perinatal exposure to the endocrine-disrupting chemical bisphenol A (BPA) exacerbated atherosclerosis in adult male PXR-humanized and ApoE KO (hPXR•ApoE−/−) mice, because BPA-induced PXR activation epigenetically regulates FA transporter CD36 expression [48]. Here, NR1I2 is linked to atherosclerosis.