ki67 and p53 were thoroughly explored by Zhai et al. [63], who reported a statistically increased number of ki67 positive cells and abnormally high expression of the oncogene p53 tumor-suppressor gene in cases of u-LMS compared with cellular and usual leiomyoma, as well as with tumors of uncertain malignant potential. The gene discussed is MKI67; the disease is leiomyoma.