Hurmath et al., 2014, reported that overexpression of IL-1β in human glioblastoma cells promoted the proliferation and migration of U87MG and U251MG cells, an event that was blocked by treating the cells with IL-1Receptor antagonist (IL-1RA) [13], resulting in inhibition of migration, invasion, and proliferative effects, indicating that the effects were mediated by IL-1β and its receptor. Here, IL1B is linked to glioblastoma.