FABP2 and Sepsis: Additionally, ghrelin has been suggested to protect against sepsis-induced intestinal injury by inhibiting ferroptosis mechanisms: By regulating oxidative stress markers, maintaining mitochondrial structural integrity, up-regulating critical tight junction proteins, and reducing serum levels of intestinal barrier injury markers, such as diamine oxidase (DAO) and fatty acid-binding protein 2 (FABP2), intestinal barrier integrity is enhanced [84].