miR-29b facilitates Th2 cytokine production and eosinophilic inflammation by targeting inducible costimulatory factors [112], while miR-15a is decreased in Th2-mediated airway inflammation, which leads to increased endothelial growth factor (VEGF) expression and promotes airway remodeling in asthma [113]. The gene discussed is VEGFA; the disease is asthma.