High copper levels can promote cell proliferation, while excessive copper exposure leads to cuproptosis.[6] For instance, bioinformatic analysis of the TCGA dataset revealed widespread upregulation of SLC31A1, which encodes a well‐known copper transport protein facilitating copper uptake across multiple cancer types,[7, 8] indicating cancer cell proliferation and growth dependent on intracellular copper. This evidence concerns the gene SLC31A1 and cancer.