During the progression of cisplatin‐induced AKI, damage to renal tubular epithelial cells causes a marked elevation in various levels of pro‐inflammatory chemokines and cytokines, namely C‐C motif chemokine ligand 2 (CCL2), C‐X‐C motif chemokine ligand 1 (CXCL1), CXCL2, and interleukin‐1 beta (IL‐1β) [4]. The gene discussed is CCL2; the disease is acute kidney injury.