In Hallmark, pathways associated with anti‐tumor immunity (inflammatory response, TNFA signaling via NFKB, IFNA response, IFNG response, and so on) were significantly activated while pathways associated with proliferation and the cell cycle (MYC targets, E2F targets, G2M checkpoint, mitotic spindle, and so on) were significantly inhibited (Figure 7G), and the same trend was observed in KEGG (Figure S10D‐E). This evidence concerns the gene MYC and neoplasm.