Studying in detail 89 genotyped patients with DCM, Augusto et al. found an RL-LGE (the “ring of fibrosis”) in 78% of patients harbouring desmoplakin (DSP) or filamin-C (FLNC) pathogenic/likely pathogenic (P/LP) variants, while the same pattern was absent in other genotypes, including titin (TTN) and sarcomeric genes [14]. The gene discussed is DSP; the disease is familial dilated cardiomyopathy.