Other genes include LMNA, linked to early-onset conduction disease and a high risk of malignant ventricular arrhythmias [31]; SCN5A, in arrhythmic phenotypes including Brugada syndrome and overlapping with DCM [29]; and RBM20 truncating variants in aggressive DCM with high arrhythmic burden [32]. This evidence concerns the gene RBM20 and familial dilated cardiomyopathy.