The observed changes in gene expression,particularly those relatedto cell growth (e.g., mitotic spindle, MYC targets, KRAS signaling,and the Verhaak-Glioblastoma Classical gene sets) and inflammatoryresponses (e.g., NFKBIA target genes, viral/inflammation gene sets),are likely on-target perturbations related to IPMK’s kinase-dependentactivity. This evidence concerns the gene IPMK and glioblastoma.