BCL2L11 and diffuse large B-cell lymphoma: In support of the latter finding as well as our recent publication demonstrating that reduced binding of the pro-apoptotic executioner BIM to BCL-2, is concurrently sequestered by the increased Mcl-1 protein in VEN-R DLBCL cells [7], our immunoprecipitation assay revealed that increased Mcl-1 could further scavenge BIM in VEN-S MOLM14 cells (Fig. 2H), thus suggesting that the increased Mcl-1 protein is phenotypically functional through the reduction of apoptosis.