UCHL1 and Parkinson disease: The link between GFAP and UCH-L1, though weaker (combined score: 0.590), suggests a potential regulatory mechanism connecting GFAP’s structural role in astrocytes to UCH-L1’s protein degradation functions, particularly in pathways like Autophagy (Reactome: HSA-9612973) and Parkinson Disease (WikiPathways: WP2371), which are relevant to protein aggregation in TBI.