As shown in Figure 3, A–E, all of these amyloidosis types could be clearly confirmed with varying relative highest abundances of TTR (ATTR1-2 to ATTR1-10; Fig. 3A), IGLL (ALλ1-2 to ALλ1-6; Fig. 3B), IGKL (ALκ1-1 to ALκ1-2; Fig. 3C), SAA (AA1-1 to AA1-3, Fig. 3D), and CALCA (ACal1-1 to ACal1-3, Fig. 3E). This evidence concerns the gene KMT2A and amyloidosis.