As outlined earlier, we were able to additionally type three samples of ALλ (CON7; lymph node (n = 1)), ATTR (CON8; bursa (n = 1)), and ALκ (CON9; myocardium (n = 1)) with unspecific or negative IHC results, but additional high abundance of ApoA4, FGA, and KRT, respectively, without defining the amyloidosis type (Supplemental Fig. S2B). This evidence concerns the gene ALK and amyloidosis.