As new biological discoveries, we found that Pph-a, which is present in the frass of P. memnon larvae fed C. paradisi leaves, inhibited the viability of MIA PaCa2, and Phe-a and Pph-a inhibited the aggregation of Aβ1–42, which is one of the causative factors in Alzheimer’s disease [21,22]. This evidence concerns the gene MEP1A and Alzheimer disease.