In heart failure, the production of proinflammatory cytokines and chemokines increases in the early stages of tissue damage, and after the rapid influx of neutrophils and monocytes into the damaged area, the influx of regulatory T cells (Tregs) and B cells and the production of anti‐inflammatory cytokines such as lipoxins, TGF‐β, and IL‐10 lead to the repair phase [52]. The gene discussed is TGFB1; the disease is heart failure.