Although no functional association is yet curated for this CKS1B pseudogene, the active expression of CKS1B has been recently linked to epithelial–mesenchymal transition (EMT) in gastric cancers [19], and the regulation of EMT by OPCML via direct interaction with AXL has been described in ovarian cancers [9]. The gene discussed is CKS1B; the disease is ovarian carcinoma.