WT1 and focal segmental glomerulosclerosis: Downregulation of WT1 through microRNA‐193a results in podocyte loss and FSGS in mice [283] and knock‐down of microRNA‐193a in human parietal epithelial cells (and hence upregulation of WT1) results in a transdifferentiation of these proliferative PECs into a non‐proliferative podocyte‐like phenotype [285], together substantiating the central role of this transcription factor for podocyte terminal differentiation.