Our present results suggest that VD enhances fatty acid oxidation via PPAR‐α in liver, which is in line with the previous study revealing that VD acts as a protective factor in non‐alcoholic fatty liver disease and that VD may alleviate hepatic steatosis via the PPAR‐α signaling pathway (Du et al. 2023). Here, PPARA is linked to metabolic dysfunction-associated steatotic liver disease.