For instance, skeletal muscle-specific miR-1 and miR-206 regulate the proliferation and differentiation of satellite cells (SCs), which function as stem cells in skeletal muscle (Chen et al, 2010); miR-206-deficient mice show delayed regeneration and an exacerbated dystrophic phenotype (Liu et al, 2012); and the inhibition of dystrophin suppressors miR-146b, miR-374a, and miR-31 has a beneficial effect on muscular dystrophy (Fiorillo et al, 2015). Here, DMD is linked to muscular dystrophy.