IUGR is associated with significant neonatal morbidity and mortality.10 Suggested pathophysiological mechanisms include glucose intolerance, insulin resistance, catabolite accumulation, and altered amino acid metabolism.11 Prenatal cardiovascular affectations of IUGR include vascular and cardiac remodelling as well as global functional impairment.12 These abnormalities may persist postnatally, leading to further compromise of an already fragile system. Here, INS is linked to fetal growth restriction.