A recent studyhas suggested the oncogenic function of OCIAD2 intumor progression, as reduced cell proliferation and migration wereobserved in HEK293 cells, a human embryonic kidney cell line, withOCIAD2 silencing. The impact of OCIAD2on cell movement was later confirmed by the wound healing assay inA549 and HCC827 lung cancer cells. Ourstudy validated the upregulation of OCIAD2 in the process of cellmotility by analyzing proteogenomics data obtained from our cohortusing MetaCore software (Figure ). This evidence concerns the gene OCIAD2 and lung carcinoma.