In a neuropathic pain model in adult mice, selective inhibition of CaMKII-positive neurons in-vivo with DREADD (engineered Designer Receptors Exclusively Activated by Designer Drugs) led to a reduction in mechanical hypersensitivity and alleviated anxiety behavior without affecting overall motor activity This critical involvement of BLA CaMKII-positive neurons was recently shown in-vivo, in paclitaxel induced neuropathic pain model in young adult mice (sex unspecified). Here, CAMK2G is linked to neuropathic pain.