Fabry disease, a lysosomal storage disorder, is caused by the absence or reduction in the activity of the alpha galactosidase A enzyme (GLA) that leads to the accumulation of glycosphingolipids (GS), in particular Globotriaosylceramide (Gb3) and a derivative, globotriaosylsphingosine (lysoGb3) (Germain, 2010). This evidence concerns the gene GLA and Fabry disease.