In contrast, the levels of Ang‐2, Pentraxin‐3, DPPIV, uPAR, vWF, C3a (all in Figure 2A), and granzyme B, galectin‐3, and SPK‐1 (data not shown) were similar in all AE subtypes and controls. This evidence concerns the gene GZMB and acrodermatitis enteropathica.