FOXM1 and Hutchinson-Gilford progeria syndrome: Additionally, Macedo et al. and Ribeiro et al. demonstrated that overexpression of only the N-terminally truncated form of FOXM1 (FOXM1-dNdK) effectively promoted cell division in fibroblast from a patient with Hutchinson-Gilford Progeria Syndrome (HGPS) (Macedo et al., 2018).