Exosomal miR-122 and miR-192 from non-alcoholic fatty liver disease (NAFLD) patient blood mimicked the palmitic acid treatment response on the Huh-7 cell exosomes, reportedly increasing COL1A1, α-SMA, and TGF-β1 levels in recipient fibroblasts [88, 89]. The gene discussed is ACTA1; the disease is metabolic dysfunction-associated steatotic liver disease.