In a silica/TGF-induced lung fibrosis model, exosomal miR-26a-5p from human umbilical cord mesenchymal stem cells (HUMSCs) disrupted a disintegrin and metalloproteinase domain-containing protein 17 (ADAM17)/neurogenic locus notch homolog protein (Notch) axis of EMT to lessen scarring in vivo (Fig. 2A-D) [41]. Here, ADAM17 is linked to pulmonary fibrosis.