In the communication with cluster 10 of monocytes, the tumor‐associated pericytes increased ECM, such as collagens and laminins, and immune‐related interactions, such as ANXA1‐FPR1 and IL1B‐CCR1, associated with the malignant behavior of gliomas [70, 71, 72, 73], and therefore suggesting a more immunosuppressive pericyte phenotype. The gene discussed is IL1B; the disease is neoplasm.