Beyond endogenous and pathogen‐derived nucleic acids, small‐molecule STING/IFN agonists—such as 5,6‐dimethylxanthenone‐4‐acetic acid (DMXAA), flavonoid compounds, and CDNs, can trigger IFN‐α/β production, thus displaying effective tumor‐suppressive activity [131, 142, 143]. Here, STING1 is linked to neoplasm.