TRPM2 and epilepsy: The results showed that compared with Trpm2fl/fl mice, the evoked AMPAR‐mediated currents instead of NMDAR‐mediated currents were increased in Trpm2fl/fl::Cx3CR1CreER mice (Figure 5G,H), providing a possible mechanism explanation for microglial TRPM2 channel increasing the neural excitability to result in epilepsy susceptibility.