While pseudo-hyperaldosteronism is typical of licorice toxicity, this patient presented with hypokalemia without alkalosis due to concurrent use of an SGLT-2 inhibitor, resulting in ketosis masking metabolic alkalosis. Clinicians should keep a high index of suspicion for licorice-induced pseudo-hyperaldosteronism, even in the absence of classical features of this condition. Here, SLC5A2 is linked to Alkalosis.