2008; Pazos et al. 1985; Pazos and Palacios 1985, 1987a, 1987b; Santana and Artigas 2017; Varnäs et al. 2004) and supporting species‐overarching inhibitory functions via 5‐HT1A receptors in areas 25 and IL. We also found a good correspondence concerning relative 5‐HT1A and 5‐HT2 distributions in the Ce and MDT, though not in the Acb of both species. Notably, deep brain stimulation of the Acb had antidepressant and antianhedonic effects in patients with treatment‐resistant depression (Bewernick et al. 2010) and in a rat model of depression (Hamani et al. 2012; Lim et al. 2015). This evidence concerns the gene HTR1A and major depressive disorder.