The complex structure of oxLDL generates particles with multivalent properties that interact with several platelet receptors, including CD36, scavenger receptor A, platelet-activating factor receptor, and lectin-like oxLDL receptor 1.9, 10, 11, 12 Among these receptors, CD36 has emerged as a prominent candidate for oxidative lipid stress–induced platelet hyperactivity through its binding to oxLDL, very low-density lipoprotein, microparticles, advanced glycation end products, S100-A9, proprotein convertase subtilisin/kexin type 9, and the COVID-19 spike protein.10 Here, CD36 is linked to COVID-19.