AKT1 and endometriosis: Considering the importance of Wnt/β-catenin and PI3K-Akt signaling pathways in the development of endometriosis, and based on our previous studies declaring the differential gene expression pattern of NE-MenSCs and E-MenSCs (10, 29), we aimed to evaluate the inhibitory effects of naringenin (an AKT inhibitor), and C-82 (a β-catenin inhibitor), as well as their simultaneous effects on improving the MenSCs from EM patients.