Moreover, decreasing fatty acid oxidation via limiting fatty acid uptake with the CD36 inhibitor, sulfo-N-succinimidyl oleate, also enhanced the recovery of cardiac function (e.g., rate pressure product) in isolated working hearts from male Wistar rats with T2D (via combination of HFD feeding plus low-dose STZ injection) subjected to hypoxia/reoxygenation [86]. This evidence concerns the gene CD36 and type 2 diabetes mellitus.