were found to be upregulated in DCM, while those of hsa-mir-30c-5p, hsa-mir-30d-5p, hsa-mir-22-3p, GSTM2, GPX3, GSTM3, ADIPOQ, HADH, PRDX2, PRDX1, SERPINA1, A2M, PRDX6, APOA1, APCS, NPPA, bilirubin, butyric acid (butyrate), alanine, glutamine, and cytidine triphosphate (ara-CTP) were found to be downregulated in DCM. This evidence concerns the gene HADH and familial dilated cardiomyopathy.