Notably, SLC1A4 and SLC1A5 demonstrate consistent upregulation, particularly in CHOL, COAD and GBM (Figure 1 (II)), whereas SLC7A10, SLC38A2, SLC38A4 and SLC38A5 exhibit tumour‐type‐specific expression patterns (Figure 1 (II)). This evidence concerns the gene SLC1A5 and glioblastoma.