These cells are a subset of γδ T cells that play a protective role in inflammation by recognizing stress‐induced molecules upregulated in immune responses to tumor cells and infections.[62] Agonists of BTN3A2 can activate the Vγ9Vδ2 T cells that provide immune surveillance and reduce neuroinflammation and tissue damage in ischemic stroke. Here, BTN3A2 is linked to neoplasm.