GPX4 and Hepatic fibrosis: This study indicates that Fx mitigates CCl4‐induced liver fibrosis in mice, likely via activation of the Nrf2/HO‐1/GPX4 axis and inhibition of ferroptosis, reflected by reduced hepatic iron overload, lipid peroxidation, and mitochondrial damage, alongside upregulated GPX4 expression (Figure 6).