Thus, we explored the biological relevance of the interactions between WAC, mTOR, and the R2TP‐TTT chaperone system by analyzing both transcriptomic and proteomic data from CPTAC (The National Cancer Institute's Clinical Proteomic Tumor Analysis Consortium) database, which integrates cancer proteogenomic data of more than 1000 treatment‐naive primary tumors from 10 cancer types [44]. The gene discussed is MTOR; the disease is neoplasm.