The list of FTH1 interactors is continually growing and includes the chemokine receptor CXCR4, whose activity is negatively modulated upon the interaction [40], the tumor suppressor p53, of which the transcriptional activity is incremented [39], the DNA replication regulatory protein NCOA4 that targets FTH1 for degradation [59], the Alacrima–Acalasia–Adrenal Insufficiency Neurological Disorder (ALADIN) protein involved in the triple A syndrome [60], and the Death Domain‐Associated nuclear protein (DAXX) [61], together with FTH1 can participate in apoptotic pathways. This evidence concerns the gene CXCR4 and triple-A syndrome.