The results of our study highlighted a dual positive effect of HE in hepatocytes in countering hepatic steatosis: on one hand, HE was able to downregulate the expression of FASN and SCD1 proteins, which are essential for the regulation of de novo hepatic lipogenesis, the metabolic pathway responsible for converting carbohydrates into fatty acids [39,40]. The gene discussed is SCD; the disease is fatty liver disease.