Furthermore, by phylogenetic analysis, the allele clustered with the African isolates, confirming that the infection was not indigenous but rather acquired in Burkina Faso, West Africa (Figs. 2 and 3). Studies from Burkina Faso have reported the presence of the RO33 (36%) and IC/3D7 (88.2%) allelic families in the msp1 and msp2 genes [4]. Here, ATAD1 is linked to infection.